Rat Neuron-specific enolase,NSE ELISA Kit

SKU:BHE10508416
Overview
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Quantitative ELISA kit for measuring rat Neuron-specific enolase (ENO2) in serum, plasma, and tissue homogenates to support metabolism studies. Sensitivity 0.156 ng/mL, detection range 0.625 ng/mL–40 ng/mL, typical assay time 1–5 h.
Target Neuron-Specific Enolase
Species Rattus norvegicus (Rat)
Sample Type(s) serum, plasma, tissue homogenates
Assay Type Sandwich ELISA (quantitative)
Sensitivity 0.156 ng/mL
Detection Range 0.625 ng/mL-40 ng/mL
Assay Time 1-5h
Options selector
Catalog no. Size
CSB-E07963r-96T 96 T
CSB-E07963r-96TX5 96 T×5
CSB-E07963r-96TX10 96 T×10
Available Options

Select from the available variant options shown for this product. Availability and lead time can vary by option.

  • Options: Size (96 T, 96 T×10, 96 T×5).
  • Lead time: options listed as "In Stock at Manufacturer" typically ship in 5–7 business days; other statuses may take longer.
  • Storage: refer to the product datasheet for storage and handling.
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Field Specification
Target Neuron-Specific Enolase
Alternative names Eno2 ELISA Kit; Eno-2 ELISA Kit; Gamma-enolase ELISA Kit; EC 4.2.1.11 ELISA Kit; 2-phospho-D-glycerate hydro-lyase ELISA Kit; Enolase 2 ELISA Kit; Neural enolase ELISA Kit; Neuron-specific enolase ELISA Kit; NSE ELISA Kit
UniProt # P07323
Species Rattus norvegicus (Rat)
Reactivity
  • Rat
Applications
  • ELISA
Assay type
  • Sandwich ELISA (quantitative)
Sample type(s) serum, plasma, tissue homogenates
Sensitivity 0.156 ng/mL
Detection range 0.625 ng/mL-40 ng/mL
Detection wavelength 450 nm
Assay time
  • 1-5h
Catalog no. (Mfr.) CSB-E07963r
Main SKU BHE10508416

Background

Neuron-specific enolase (ENO2) is a biological molecule commonly studied in metabolism research. It is commonly used as a molecular readout in mechanistic and biomarker-focused studies.

UniProt: P07323

Biological context

Researchers often monitor Neuron-specific enolase in serum, plasma, and tissue homogenates to better understand themes such as energy homeostasis, glucose and lipid metabolism, and insulin sensitivity and endocrine regulation. In many model systems, measured levels can shift with physiology, experimental perturbation, or disease-associated changes, making careful biological interpretation important.

Interpreting changes in measured levels

Depending on sample matrix and study design, increases or decreases in Neuron-specific enolase may reflect differences in expression, secretion, turnover, or compartmentalization rather than a single mechanism. Interpretation is typically strengthened by evaluating related molecules (for example, insulin, adipokines, lipid-transport proteins, and stress-related enzymes) and by keeping pre-analytical variables consistent across groups.

Nomenclature

In publications and databases, Neuron-specific enolase may also appear under names such as Eno2 and Eno-2. When comparing studies, confirm that the reported analyte refers to the same molecule and species context.

Why ELISA data are widely used

ELISA is a common approach for quantitative measurement of proteins and biomarkers in complex samples, enabling comparisons across experimental groups and time points. When integrating results with other readouts, consider species biology, sample type, and the broader pathway context that Neuron-specific enolase participates in.

Can’t Find What You’re Looking For? We can help you source the best match or customize an ELISA solution for your study. Options may include alternative target synonyms, different species reactivity, sample type/matrix compatibility (serum/plasma/lysate/supernatant), assay format (sandwich/competitive), sensitivity/range, detection chemistry (colorimetric/fluorescent/chemiluminescent), plate format (pre-coated/uncoated, strips vs full plate), and bulk or custom packaging. Click Talk to a Scientist to submit a request form, email us at support@biohippo.com, or explore our Research Services for additional support. Our team will be in contact with you shortly.

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Magnetic resonance spectroscopy for assessment of brain injury in the rat model ofSepsis

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Dexmedetomidine reduces the neuronal apoptosis related to cardiopulmonary bypass by inhibiting activation of the JAK2-STAT3 pathway

Chen Y.et al,Drug Des Devel Ther,2017

Hypothermia followed by rapid rewarming exacerbates ischemia-induced brain injury andAugments inflammatory response in rats.

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Endotoxin-induced lung alveolar cell injury causes brain cell damage

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Interrupted intracarotid artery cold saline infusion as an alternative method for neuroprotection after ischemic stroke

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